Peripheral Neuropathy Treatment Success

Today we are here with Amir, he came intoour office Valentine Chiropractic originally by iving by and seeing our our previousadsthe newspaper regarding Laser and Decompression treatments. He camewith Plantar Fascitisand came to realize after being examined that he really had Peripheral Neuropathy. He wentto Kaiser, tried to put hima cast for 4 weeks and he started to lose strength inhis legs, he was also on pain pills which were making him violently sick. He was lookingfor some other modality to help him, he felt there had to be something out there that couldhelp. I happened to be iving by your building and i looked and saw that you talked aboutbeing Gluten Free and Peripheral Neuropathy,

so I called and talked to your brother Leonard Valentine and had a good conversation and felt very comfortable that you guys couldhelp me with my chronic plantar fascitis. The pain I had was not justmy heels it wasthe whole bottom of my foot hurt plus my heel and Kaiser was telling me it could come fromyour back so they did back xrays, nothing showed up on my back xrays, so then theysaid it was my weight. It turns out it wasn't my weight it turns out I had another ailment,that needed to be treated and healed, so now since starting treatment which has been about2 weeks and the whole bottom of my foot and my heel that I'm not feeling any pain in.So this is the first time2 years that

you have gone 2 weeks without any pain atall? Correctthat's is phenomenal and being off pain pills for a month, that has beenthe first time2 years. What would you say to those people all around the world whosuffer with plantar fascitis or peripheral neuropathy? We work with people all arounddifferent states who suffer with this very same problem, we can help those people aslong as they are willing to reach out. What would you say to the people else where sufferingwith Peripheral NeuropathyI would say this program works, I haven't even beenthisprogram 7 weeks, maybe 57 weeks this has got to be the program that is going to helppeople, I have found nothing else to help

meyour program is wonderful for treatingand fixing people with Plantar Fascitis or Peripheral Neuropathy.

Diabetic nephropathy al presentation treatment

Diabetic nephropathyis one of the most common and serious chronic compliions associated with diabetes mellitus. In this tutorial, let'sdiscuss how the mechanisms underlying diabetic nephropathy correlate with the al presentation as well as the treatment of the disease. Now fortunately the mechanisms

underlying diabetic nephropathy, directly correlate withthe al presentation. And the first alfinding of the disease is somewhat paradoxicallyan increased kidney filtration rate orglomerular filtration rate. So, diabetic nephropathy,if you break down the term into nephro and pathy literally means kidneydisease caused by diabetes.

Now typically kidney disease is marked by a decreased filtration rate, so why is it that the first al stage of diabetic nephropathy is that of an increased glomerularfiltration rate? Well recall that the earliest mechanism contributing to diabetic nephropathy is an increased pressurestate, over hereblue.

And this is due to hypertension and efferent vasoconstriction. So let's use a common garden hose to help illustrate how thisincreased pressure state will ultimately resultan increased glomerular filtration rate. So, imagine you have this garden hose and it has a small holein the middle of it.

So first you're gonna open up the spigot and increase the pressureand flow through the hose. Intuitively, this isgoing to increase the rate at which water is leakingfrom the holethe hose. Next, you partially kink off the end of the hose distal to the hole, and once again this isgonna further increase the rate at which waterleaks from the hose.

This is essentially what'soccurringthe glomerulus with the hypertension representing the opening up of the spigot and increasing the pressurebefore the glomerulus,front of the glomerulus, and the efferent vasoconstriction representing the kinking off of the hose, which causes this back pressure.

Diabetic nephropathy Mechanisms

One of the most seriouschronic compliions of diabetes mellitus is a condition known as diabetic nephropathy. Which, if you break down the term into nephro and pathy literally means kidney disease that occurssecondary to diabetes. And it's actually pretty common as it eventually affects about20% to 40% of all individuals

with diabetes, includingboth type I and type II. In this tutorial, let'stalk about the mechanism underlying the causeof diabetic nephropathy and how individuals withdiabetes develop the condition. So diabetic nephropathyis a chronic compliion of diabetes mellitus. Meaning, it usually has aslow progression over decades after the initial diagnosis of diabetes.

And to give you anoverview of what happens, an insulin deficiency due to the diabetes resultshyperglycemia, which then causes hypertensionand kidney dysfunction. This kidney function isactually then further worsened by the hypertension. And ultimately, all of thisresultskidney failure, which can have very severeand potentially even

life threateningcompliions, such as anemia, electrolyte imbalances,such as metabolic acidosis, and heart arrhythmias. Now, before we dive into the mechanism of diabetic nephropathy, let'sbriefly the structure of the glomerulusthe kidney, by bringinga diagram here. So, the glomerulus isthe portion of the kidney

where blood is initially filtered. So blood enters the glomerulus over here, through this afferent arterial, and then leaves the glomerulus throughthe efferent arterial. And you can remember this, that it leaves throughthe efferent arterial for E for exit, or efferent. And while the blood iswithin the glomerulus,

there's this advanced filtration system, which we'll talk about morea minute. And the filtered fluidthat exits the blood is known as a filtrate and it collectsBowman's space before it enters into thetubules of the nephron where further reabsorptionand secretion occurs before it exits the kidneyinto the ureters as urine.